Fertility could be regained if those 2 ends could be rejoined such that sperm can once again pass through the vas unhindered. However, many factors dictate whether or not this actually happens including the distance between the 2 ends of the vas, the stitches used to seal up the ends of the vas and infections and scar tissue formation after the original vasectomy operation. The success rate is therefore low.
The actual operation is usually done as a day case but under general anaesthesia. Incisions are made to pull out the ends of the vas deferens, the stitches removed, the 2 ends prepared and then stitched together. The skin wounds are then stitched.
The typical vasectomy reversal procedure is called a “Vasovasostomy.” It is performed with a microsurgical technique to reconnect the previously cut ends of the vas deferens, to open the vas channel to restore fertility.
Occasionally, the original vasectomy may cause an increase in pressure at the epididymis, a special tube upstream from the blocked end of the vas deferens, resulting in a “blow-out” or blockage. This should be bypassed. The surgeon does this by reconnecting the vas end to the epididymis even further upstream from the blockage. This more complicated reversal procedure is known as a “Vasoepididymostomy.
With the patient under anaesthesia, a 1-2 inch incision is made in the scrotal skin over the old vasectomy site. The two ends of the vas deferens are found and freed from the surrounding scar tissue. A drop of fluid from the testicular end of the vas is placed on a glass slide and examined using a light microscope. This is a crucial part of the operation because the information obtained is used to decide what type of microsurgical reconstruction needs to be performed. Since the testicle continues to produce sperm after a vasectomy, the fluid in the vas should contain sperm.
3 possible scenarios may be encountered when examining the vassal fluid. The first and best scenario is that the vast fluid contains whole sperm. The second possible finding is that the fluid is thin and copious and contains only sperm parts or no sperm. The third is that the fluid is thick, and pasty and contains no sperm. This last scenario usually means that a “blowout” or rupture has occurred in the epididymis. Sperm leaks out if the pressure in the tubule becomes greater than the resistance in the wall of the tubule, similar to the way a pipe breaks in the basement when the water pressure gets too high. The body tries to heal this tubule and the scar forms. This causes a second blockage in the epididymis, which needs to be bypassed to allow the sperm to get out into the vas. If this second blockage is present and is not recognized then the operation is doomed to failure.
After a vasectomy, the testes remain in the scrotum where Leydig cells continue to produce testosterone and other male hormones that continue to be secreted into the bloodstream. Some studies find that sexual desire is unaffected in over 90% of vasectomized men, whereas other studies find higher rates of diminished sexual desire, for example nearly 20%. The sperm-filled fluid from the testes contributes about 10% to the volume of ejaculation (in men who are not vasectomized) and does not significantly affect the appearance, taste, texture, or smell of the ejaculation.
When the vasectomy is complete, sperm cannot exit the body through the penis. Sperm is still produced by the testicles, but they are broken down and absorbed by the body. Much fluid content is absorbed by membranes in the epididymis, and much solid content is broken down by the responding macrophages and re-absorbed via the bloodstream. Sperm is matured in the epididymis for about a month once it leaves the testicles. Approximately 50% of the sperm produced never make it to the orgasmic stage in a non-vasectomized man. After a vasectomy, the membranes increase in size to absorb and store more fluid; this triggering of the immune system causes more macrophages to be recruited to break down and re-absorb more of the solid content. Within one year after a vasectomy, sixty to seventy per cent of vasectomized men develop anti-sperm antibodies. In some cases, vasitis nodosa, a benign proliferation of the ductular epithelium, can also result.
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